In our study, we compared the levels of this biomarker in samples from other tauopathies and Parkinson’s disease, confirming that the changes in miR-519a-3p are specific to Alzheimer’s disease,” said IBEC senior researcher Rosalina Gavín, UB associate professor and co-leader of the study.

coooool!

This is important because because it's easily measurable, comparable, and likely to be unique. This was not the case with the Amyloid Plaque route of research (recently announced to be retracted due to falsified imaging in the original studies) where it could exist in non-Alzheimer patients in similarly measurable quantities. Takes a lot of the guesswork out.

Science fucking rules.