Have fun with your eventual colon cancer.
Do you have non-observational data to support this position that the lack of fibre is casual for colon cancer?
You were removed from the community 8 months ago - https://discuss.online/modlog?page=1&actionType=All&userId=2971197
At the time the automation identified your user behavior as inorganic.

but mistakes can happen, perhaps the script was in error. would you like to be added back into the community?
Here is the guiding moderation philosophy of that community: https://hackertalks.com/post/13655318
Maybe they should get a little more fiber in their diet.
Fibre is not an essential nutrient, there is no need to consume it - especially if your not eating carbohydrates and producing your own short chain fatty acids.
https://en.wikipedia.org/wiki/Mirepoix
mixture of diced vegetables cooked with fat (usually butter) for a long time on low heat without colouring or browning. The ingredients are not sautéed or otherwise hard-cooked, because the intention is to sweeten rather than caramelise them.
Vegetable pemmican?
Wonderful collagen!

Yes, some celery in the broth... still getting everyone onboard with no-plants. The person who added the celery into the broth doesn't even like celery, they were just following a guide. Baby steps.
After more than ten years of treating patients with a carnivore diet and studying the relationship between diet and chronic disease, I have learned that most of what we are told about healthy eating is not only wrong but is actively making people sicker.
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Core protocol
Margins and re-exposure
Why the diet works
Disease mechanisms and examples
Species-specific diet logic
Practical finish
References
Does carnivore make you stronger or weaker? I hear all the time that you need carbs to perform at your best athletically. Is that actually true, or have we been misinformed?
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Low-carb athletic performance
Fat adaptation and the crossover point
Hydration, electrolytes, and bicarbonate
Meal timing and blood glucose
Training boundaries
References
Diet has a huge impact on the brain and mood.
There is lots of research coming out on the metabolic brain connection, basically try a well formulated ketogenic diet for a few weeks and see if you feel better, keep a daily mood journal!. Many, many, many people do, especially around anxiety symptoms. See the metabolic mind organization for the current research results.
One warning if your taking medication: changing your metabolism can make medicine much stronger then anticipated, so work with your doctor to modify prescriptions as needed.
$2000 for a phone is a big ask, unlock the bootloader and I'll think about it
Dr. Eric Westman sits down with Craig Emmerich, co-author of The Art of Metabolic Health, to discuss why metabolic health is often misunderstood by modern medicine and how low carb, keto, carnivore, protein, and fat-burning can change the way we think about chronic disease. Craig shares how his wife Maria’s health journey led them into the low carb world, why doctors still often fail to ask about food, how the body prioritizes alcohol, glucose, fat, and protein, and why ketosis is a normal metabolic state rather than something to fear. They also discuss dementia, kidney health, coffee, children’s nutrition, protein, and the power of real food in helping people take back their health.
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Origin and background
Purpose of the new book
Fuel priority and ketosis
Clinical signals and research gaps
Families, children, and friction points
References
The next research question is : how would a high carb diet with sglt2 inhibitors compare against a ketogenic diet without sglt2 inhibitors in IHF? i.e. are the benefits from the slgt2 based on ketone metabolism? we don't know. The east-asian's might also have non-impaired ketogenic metabolism without the sglt2s.
Mendelian randomization (hypothesis generating) of epidemiology (a systematic review of epidemiology is still only as strong as the epidemiology itself, so not at all) in patients with heart failure on sglt2's. Lower ketone bodies had better HF outcomes, but also along ethnic lines (so other variables could account for it as well)
Not that these populations were not doing ketogenic eating patterns, or low carb, just their background ketone levels in their typical diet while on sglt2s, so high carb
The MR finding that genetically elevated ketones causally increase HF risk challenges the “ketones-as-superfuel” hypothesis and suggests potential harm from chronic ketone elevation. This aligns with emerging data from genetic mouse models showing that constitutive ketone overproduction or impaired use leads to cardiac dysfunction.
But that doesn't follow also from the paper
Ketone metabolism dysfunction was defined as β-HB levels in the highest tertile (>2.41 mmol/L) combined with acetoacetate:β-HB ratio <0.15, indicative of impaired ketone use.
SGLT2 inhibitors promote endogenous ketogenesis through multiple mechanisms including hepatic substrate availability modification and direct metabolic effects, potentially contributing to their cardioprotective properties.
So its possible the sglt2 is interfering with ketone use (hence the ketone build up in a high carb diet), resulting in their negative outcomes, but their opinion is against ketones in general.
Even if you take this epidemiology as truth - are you a east asian taking a sglt2 alongside a high carb diet while maintaining ketone levels > 2.25mmol/l?
Background - Heart failure with ischemic cause is associated with substantial cardiovascular mortality. SGLT2 (sodium glucose cotransporter 2) inhibitors demonstrate cardiovascular benefits, but interindividual response variability remains poorly understood. We investigated the relationship between baseline ketone body metabolism and SGLT2 inhibitor response in heart failure with ischemic cause across diverse genetic backgrounds.
Methods - We analyzed metabolomics data from 3847 patients with heart failure with ischemic cause across 23 countries (2020–2024). Ketone body metabolites (β‐hydroxybutyrate, acetoacetate, acetone) were quantified by liquid chromatography‐mass spectrometry. SGLT2 inhibitor response was assessed via a composite end point including cardiovascular mortality, heart failure hospitalization, and kidney function decline. Analyses included multivariate Cox regression, machine learning (Random Forest, Extreme Gradient Boosting), and Mendelian randomization, integrating Global Burden of Disease 2021 data across 5 genetic ancestry groups.
Results -Baseline β‐hydroxybutyrate inversely correlated with SGLT2 inhibitor outcomes (r=−0.67, P<0.001). The lowest ketone tertile demonstrated superior outcomes (hazard ratio [HR], 0.58 [95% CI, 0.51–0.66], P<0.001), and the highest tertile showed elevated risk (HR, 1.58 [95% CI, 1.39–1.79], P<0.001). East Asian populations exhibited 34.24% higher baseline ketone levels (2.47±0.83 versus 1.84±0.61 mmol/L, P<0.001) with attenuated treatment benefit versus European ancestry. Machine learning models achieved area under the receiver operating characteristic curve of 0.8245 (95% CI, 0.8012–0.8478) predicting individual outcomes from baseline metabolomic profiles.
Conclusions - Baseline ketone body metabolism is strongly associated with SGLT2 inhibitor outcomes in heart failure with ischemic cause, with marked interancestry variability. Metabolomic profiling may inform precision medicine approaches to therapeutic decision‐making, pending prospective validation.
Full Paper - https://doi.org/10.1161/JAHA.125.048427
When traveling I usually get two esims from different providers, for some level of redundancy
David Baszucki - founder and CEO of Roblox - sits down with his son Matthew for the first time on a podcast together. They walk through Matthew's five-year journey with severe, treatment-resistant bipolar I disorder: the first manic episode in 2016, multiple hospitalisations, the dozens of medications, the 2017 incident in Los Angeles when David flew down on a rescue mission to find Matt homeless on the streets, and the discovery by Matthew’s mother Jan of ketogenic therapy that sent Matt's symptoms into remission. David and Matthew talk about how their family journey led to the establishment of the rapidly growing field of metabolic psychiatry their family now funds, and the daily metabolic practices both father and son use today.
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Family crisis and biology
Keto turning point
Care-system gaps
Metabolic model
Research path
Daily protocol
Advice and cautions
References
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Using a bioenergetic approach and physiological, anatomical, archaeological, ethnographic, isotopic, botanical, genetic and zoological evidence my research has identified an obligated animal fat requirement in human nutrition beginning with the Homo erectus. In a recent paper published PLoS ONE (linked in bio), written in co-operation with researchers from Tel Aviv University: we applied a bioenergetic model to test the hypothesis that shortage of animal fat that developed locally in the Levant 400 thousand years ago due to the disappearance of elephants was an important factor in the evolution of a new modern human lineage. Presently, we continue with the application of the Obligated Fat Model in an attempt to understand more recent critical developments in humans' existence. Some of our present research results will be reported.
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Man the fat hunter
Fat as the real target
Obligatory fat and prey size
Elephants, deer, and the Levantine shift
Prime-age hunting as the fat solution
Brains, tracking, and Homo sapiens
Refining the expensive-tissue idea
References
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Here is the paper they published - https://doi.org/10.1371/journal.pone.0028689
Sharing my carnivore journey as an ex-vegan of 14-years who developed ulcerative colitis while vegan and only got it into permanent, natural remission once I started the carnivore diet.
She does have a more recent update, but google age restricted it..... that is curious.
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Background and scope
Ulcerative colitis and digestion
Skin, complexion, and inflammation signals
Mental state, focus, and daily functioning
Body composition, cravings, and PMS
Hair, nails, skin density, and collagen
Teeth, mouth feel, and animal fat
Current limits and negatives
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Dr. Eric Westman (Duke University) walks through his clinical perspective on low-carb and ketogenic dieting, focusing on how he thinks carbohydrates relate to obesity, diabetes risk, and body composition. He describes the approach he teaches patients, the “why” behind his food lists, and the behavioral hurdles he says often derail adherence. Along the way, he contrasts mainstream nutrition messaging with what he reports seeing in clinic, and he shares practical examples meant to make the framework easy to follow.
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Central problem
Low-carb history
What changed the old training
Food as a human recipe
Essential carbohydrate and nutrition
Obesity clinic application
Fat burning made simple
Communication and regulation
Final clinical recipe
References
In this episode of The Feldman Protocol, Dave Feldman sits down with Peter Ballerstedt (PhD) — forage agronomist and founder of Grass Based Health.
The first 10 minutes are a great discussion of institutional censorship! We need to replace youtube with a open federated distribution model.
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Suppression and stakes
Grass-Based Health and ruminant biology
Protein quality and amino acids
Processing, plant protein, and DIAAS
Food-policy origins
Land, ecology, and farming systems
Biomass and feed-food competition
Animal-source foods, development, and adequacy
Protein share, anti-nutrients, and nutrient response
Guidelines, ethics, and study limits
Medical institutions and carbohydrate reduction
Ruminants, keto demand, and animal fat
AI, education, and institutional knowledge
Closing health and human needs
References
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Salt Beef as Everyday Preservation
How Salt Keeps Meat Usable
Salt Beef Compared with Salt Pork
Cooking Salt Beef
Shipboard Handling
Military Supply Scale
Making Salt Beef
Using Salt Beef
Taste Test
References
We tackle the common 'anti-carnivore debate' arguments head-on, questioning the logic behind comparing human anatomy to that of carnivores. This educational video uses science to debunk claims, highlighting why such comparisons are flawed. Join us as we discuss nutrition from a carnivore perspective and address propaganda with facts.
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Fangs and claws
Human gut and stomach signals
Nutrient requirements
Survival versus thriving
Human advantage
References
It looks interesting, thanks for the tip!
Suet is the preferred fat for making pemmican!